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Esterization of the testosterone molecules provides for a sustained (but non-linear) release of testosterone from the injection depot into the blood plasma. The subsequent rise in LH and estradiol concentrations occurs, then the remaining testosterone is slowly absorbed by the liver. The increased estrogen content is not entirely due to the effect of SDF8, since the increased estrogen can also contribute to increased production of testosterone from SDF8. The addition of the estrogenic effects of SDF8, in part, to a testosterone administration with minimal adverse effects (such as increased cortisol release from the adrenal cortex) results in a prolonged increase in testosterone levels in the blood and an increased plasma estradiol concentration, which may be responsible for the increase in estradiol production seen in female dogs. Furthermore, it appears that the combination of SDF8 and testosterone can increase the level of endogenous sex steroids in a manner similar to the increase in cortisol production by a hormone-rich diet. The overall effect of increasing testosterone levels (with an increased level of plasma estradiol) in females appears to promote normal estradiol secretion in the pituitary and ovaries, while at the same time lowering the levels of prolactin in the adrenal pituitary, thereby maintaining the normal function of those glands. These changes appear to be related in part to an increased production of testosterone from the pituitary and the stimulation of a progesterone production from the ovaries, and thus increase the activity/production of estrogen. Related Article: